Definition And Classification

Disease Overview

  • Hyper-responsive pulmonary syndrome secondary trapped microfilariae within lung tissue.
  • Clinical manifestation lymphatic filariasis.
  • Classified extrinsic pulmonary eosinophilic syndrome.

Etiology And Vectors

Causative Organisms

  • Nematodes: Wuchereria bancrofti, Brugia malayi, Brugia timori.
  • Unusual filariasis form occasionally involving Dirofilaria imitis.

Vectors

  • Transmitted mosquito bites serving vectors.
  • W. bancrofti vectors: Culex, Anopheles, Aedes mosquitoes.
  • Predominantly filarial endemic regions (Indian subcontinent, South East Asia, South America, Africa).
  • Higher incidence nonimmune individuals travelling endemic regions compared permanent residents possessing developed immunity.

Pathophysiology

Immune Response Cascade

  • Type 1 hypersensitivity reaction microfilariae trapped pulmonary microcirculation, lymphatic system, bloodstream.
  • Microfilariae released periodic lymphatic system.
  • Trapped microcirculation triggers immune response releasing eosinophils.
  • Eosinophils central pathogenesis.
  • Eosinophil degranulation releases Eosinophilic Cationic Protein (ECP), Eosinophil-Derived Neurotoxin (EDN), Major Basic Proteins (MBP), Eosinophil Peroxidase (EPO).
  • Clearance microfilariae simultaneous lung damage.
  • Complement activation, opsonization antifilarial antibodies facilitate microfilariae clearance.

Airway Hyperactivity

  • MBP-2 associated airway hyperactivity.
  • Interleukin-4 (IL-4) induces airway hyperactivity.
  • Interferon-gamma (IFN-gamma) suppresses airway hyperactivity.
  • Generates overactive systemic, pulmonary Th2 response.
  • Massive pulmonary eosinophilia, increased IL-4, IL-5, filarial-specific Immunoglobulin G (IgG), IgM, IgE antibodies.

Histopathology

Temporal Disease Progression

Disease StageMicroscopic Findings
EarlyHistiocyte infiltration lung parenchyma causing initial symptoms.
Sub-AcuteEosinophilic interstitial infiltration. Progression eosinophilic abscesses, eosinophilic granulomas, eosinophilic bronchopneumonia.
6 Months To 2 YearsMixed cell reaction including histiocytes, eosinophils, epithelioid cells, lymphocytes.
UntreatedPulmonary fibrosis.

Clinical Features

Systemic And Pulmonary Manifestations

  • Slow onset.
  • Dry cough, paroxysmal, nocturnal,.
  • Dyspnea, wheezing,.
  • Systemic symptoms: Fever, malaise, anorexia, weight loss.
  • Chest auscultation reveals wheezing, crepitations.

Extrapulmonary Manifestations

  • Lymphadenopathy,.
  • Hepatosplenomegaly.
  • Organomegaly abdominal palpation.

Evaluation And Diagnosis

Investigations

InvestigationCharacteristic Findings
Complete Blood CountLeukocytosis, peripheral blood eosinophilia >3,000/mm3,.
Serum ImmunoglobulinElevated quantitative IgE level,.
Indirect ELISAElevated filarial antibody titers,.
Stool ExaminationRule out alternate parasites causing pulmonary eosinophilia.
Chest X-RayReticulonodular opacities, miliary mottling middle/lower zones. Snowflake-like appearance. Normal 20-30% cases.
Chest CT ScanBronchiectasis, lymphadenopathy, pleural effusion (utilized unestablished diagnosis).
Pulmonary Function TestMixed pattern, predominant restrictive, mild-to-moderate obstructive.

Management

Pharmacotherapy

  • Diethylcarbamazine (DEC) 6 mg/kg 21 days.
  • Alternative DEC dosing: 10 mg/kg three divided doses 3 weeks (requires 2-3 spaced courses occasionally).
  • Corticosteroids concomitant therapy reduces airway inflammation chronic setting.
  • Ivermectin targets microfilariae.
  • Albendazole targets adult worms.

Complications And Differential Diagnosis

Complications Untreated Disease

  • Pulmonary fibrosis.
  • Chronic bronchitis, chronic respiratory failure.
  • Pulmonary hypertension, cor pulmonale secondary destructed microfilariae embolization lung capillaries.

Differential Diagnosis

  • Infectious Eosinophilia: Strongyloides, Toxocara, Ascariasis.
  • Respiratory: Bronchial asthma, Allergic Bronchopulmonary Aspergillosis (ABPA), Allergic rhinitis,.
  • Eosinophilic Syndromes: Acute/chronic eosinophilic pneumonia, Churg–Strauss syndrome, DRESS syndrome.
  • Infections: Miliary tuberculosis, Fungal pneumonia.